A novel F8 -/- rat as a translational model of human hemophilia A

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A novel F8 -/- rat as a translational model of human hemophilia A. / Nielsen, L. N.; Wiinberg, B.; Häger, M.; Holmberg, H. L.; Hansen, J. J.; Roepstorff, K.; Tranholm, M.

I: Journal of Thrombosis and Haemostasis, Bind 12, Nr. 8, 08.2014, s. 1274-1282.

Publikation: Bidrag til tidsskriftTidsskriftartikelForskningfagfællebedømt

Harvard

Nielsen, LN, Wiinberg, B, Häger, M, Holmberg, HL, Hansen, JJ, Roepstorff, K & Tranholm, M 2014, 'A novel F8 -/- rat as a translational model of human hemophilia A', Journal of Thrombosis and Haemostasis, bind 12, nr. 8, s. 1274-1282. https://doi.org/10.1111/jth.12635

APA

Nielsen, L. N., Wiinberg, B., Häger, M., Holmberg, H. L., Hansen, J. J., Roepstorff, K., & Tranholm, M. (2014). A novel F8 -/- rat as a translational model of human hemophilia A. Journal of Thrombosis and Haemostasis, 12(8), 1274-1282. https://doi.org/10.1111/jth.12635

Vancouver

Nielsen LN, Wiinberg B, Häger M, Holmberg HL, Hansen JJ, Roepstorff K o.a. A novel F8 -/- rat as a translational model of human hemophilia A. Journal of Thrombosis and Haemostasis. 2014 aug.;12(8):1274-1282. https://doi.org/10.1111/jth.12635

Author

Nielsen, L. N. ; Wiinberg, B. ; Häger, M. ; Holmberg, H. L. ; Hansen, J. J. ; Roepstorff, K. ; Tranholm, M. / A novel F8 -/- rat as a translational model of human hemophilia A. I: Journal of Thrombosis and Haemostasis. 2014 ; Bind 12, Nr. 8. s. 1274-1282.

Bibtex

@article{137a5e68c05f4e688f6a9fc6dc26a7ef,
title = "A novel F8 -/- rat as a translational model of human hemophilia A",
abstract = "Background: In preclinical hemophilia research, an animal model that reflects both the phenotype and the pathology of the disease is needed. Objectives: Here, we describe the generation and characterization of a novel genetically engineered F8-/- rat model. Methods: The rats were produced on a Sprague Dawley background with the zinc finger nuclease technique. A founder with a 13-bp deletion in exon 16 causing a premature translational stop in the C-terminal part of the A3 domain of factor VIII was selected, and a breeding colony was established. Results: Seventy per cent of the homozygous rats had clinically manifest spontaneous hemorrhagic episodes that needed treatment. The F8-/- rats had no detectable FVIII activity, and had a significantly prolonged activated partial thromboplastin time (APTT) and clot formation time as compared with wild-type (WT)/WT rats. In vitro spiking of rat plasma with human recombinant FVIII resulted in dose-dependent normalization of the APTT. Conclusion: On the basis of the targeted deletion in F8, and the distinct physical and analytic characteristics of the rat, we conclude that an FVIII-deficient rat strain has been generated that has the potential to contribute greatly to translational research.",
keywords = "Animal model, Gene knockout, Hemophilia, Phenotype, Rattus",
author = "Nielsen, {L. N.} and B. Wiinberg and M. H{\"a}ger and Holmberg, {H. L.} and Hansen, {J. J.} and K. Roepstorff and M. Tranholm",
year = "2014",
month = aug,
doi = "10.1111/jth.12635",
language = "English",
volume = "12",
pages = "1274--1282",
journal = "Journal of Thrombosis and Haemostasis",
issn = "1538-7933",
publisher = "Wiley-Blackwell",
number = "8",

}

RIS

TY - JOUR

T1 - A novel F8 -/- rat as a translational model of human hemophilia A

AU - Nielsen, L. N.

AU - Wiinberg, B.

AU - Häger, M.

AU - Holmberg, H. L.

AU - Hansen, J. J.

AU - Roepstorff, K.

AU - Tranholm, M.

PY - 2014/8

Y1 - 2014/8

N2 - Background: In preclinical hemophilia research, an animal model that reflects both the phenotype and the pathology of the disease is needed. Objectives: Here, we describe the generation and characterization of a novel genetically engineered F8-/- rat model. Methods: The rats were produced on a Sprague Dawley background with the zinc finger nuclease technique. A founder with a 13-bp deletion in exon 16 causing a premature translational stop in the C-terminal part of the A3 domain of factor VIII was selected, and a breeding colony was established. Results: Seventy per cent of the homozygous rats had clinically manifest spontaneous hemorrhagic episodes that needed treatment. The F8-/- rats had no detectable FVIII activity, and had a significantly prolonged activated partial thromboplastin time (APTT) and clot formation time as compared with wild-type (WT)/WT rats. In vitro spiking of rat plasma with human recombinant FVIII resulted in dose-dependent normalization of the APTT. Conclusion: On the basis of the targeted deletion in F8, and the distinct physical and analytic characteristics of the rat, we conclude that an FVIII-deficient rat strain has been generated that has the potential to contribute greatly to translational research.

AB - Background: In preclinical hemophilia research, an animal model that reflects both the phenotype and the pathology of the disease is needed. Objectives: Here, we describe the generation and characterization of a novel genetically engineered F8-/- rat model. Methods: The rats were produced on a Sprague Dawley background with the zinc finger nuclease technique. A founder with a 13-bp deletion in exon 16 causing a premature translational stop in the C-terminal part of the A3 domain of factor VIII was selected, and a breeding colony was established. Results: Seventy per cent of the homozygous rats had clinically manifest spontaneous hemorrhagic episodes that needed treatment. The F8-/- rats had no detectable FVIII activity, and had a significantly prolonged activated partial thromboplastin time (APTT) and clot formation time as compared with wild-type (WT)/WT rats. In vitro spiking of rat plasma with human recombinant FVIII resulted in dose-dependent normalization of the APTT. Conclusion: On the basis of the targeted deletion in F8, and the distinct physical and analytic characteristics of the rat, we conclude that an FVIII-deficient rat strain has been generated that has the potential to contribute greatly to translational research.

KW - Animal model

KW - Gene knockout

KW - Hemophilia

KW - Phenotype

KW - Rattus

UR - http://www.scopus.com/inward/record.url?scp=84905657066&partnerID=8YFLogxK

U2 - 10.1111/jth.12635

DO - 10.1111/jth.12635

M3 - Journal article

C2 - 24931420

AN - SCOPUS:84905657066

VL - 12

SP - 1274

EP - 1282

JO - Journal of Thrombosis and Haemostasis

JF - Journal of Thrombosis and Haemostasis

SN - 1538-7933

IS - 8

ER -

ID: 345682769